Description: Pathogenicity of Human Herpesviruses due to Specific Pathogenicity Genes Please note: this item is printed on demand and will take extra time before it can be dispatched to you (up to 20 working days). Author(s): Yechiel Becker, Gholamreza Darai Format: Paperback Publisher: Springer-Verlag Berlin and Heidelberg GmbH & Co. KG, Germany Imprint: Springer-Verlag Berlin and Heidelberg GmbH & Co. K ISBN-13: 9783642850066, 978-3642850066 Synopsis Six members of the Herpesviridae family are human pathogens, including herpes and 2 (HSV-I and 2), Epstein-Barr virus (EBV), varicella zoster simplex virus I virus (VZV), human cytomegalovirus (HCMV), and human herpesvirus 6 (HHV 6). Each of these viruses is capable of causing distinct diseases of varying severity in children, young adults, and the aged. The diseases range from infection of epithelial tissue to the infection of internal organs and white blood cells. A common feature of the six pathogenic human herpesviruses is their ability to latently infect different cell types in which the viral DNA is not integrated and is unable to express its pathogenicity. Reactivation of the herpesviruses is a result of cellular processes which reactivate viral genes, leading to virus progeny and to signs of infection. Due to their ability to become latent after initial infection, once the pathogenic herpesviruses infect children they are maintained throughout life, having the potential of cause various diseases upon reactivation.
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Location: Aldershot
End Time: 2025-01-27T12:04:28.000Z
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Book Title: Pathogenicity of Human Herpesviruses due to Specific Pathogeni...
Number of Pages: 387 Pages
Publication Name: Pathogenicity of Human Herpesviruses Due to Specific Pathogenicity Genes
Language: English
Publisher: Springer-Verlag Berlin AND Heidelberg Gmbh & Co. KG
Item Height: 235 mm
Subject: Medicine, Chemistry, Biology
Publication Year: 2011
Type: Textbook
Item Weight: 616 g
Author: Yechiel Becker, Gholamreza Darai
Item Width: 155 mm
Series: Frontiers of Virology
Format: Paperback
